Wednesday, September 30, 2009

Treatment and CAM for Sky-Attic-Kah

Conservative Treatment

Conservative treatment is aimed at pain reduction. Initial treatment starts with cold packs or heat, NSAIDs (eg,Celebrex, Ponstan, Motrin), muscle relaxants, and pain medications.

Education about proper mechanics (bending and lifting), physical therapy, and epidural steroid injections may also help patients return to full activity.

Surgical Treatment

Some patients do not respond to nonsurgical care and experience persistent disabling sciatica. These patients may benefit from surgery.

The role of surgery is to remove the disc herniation or stenosis (narrowing of canal) that is pressing on the affected lumbar nerve to ease the leg pain and associated symptoms of numbness and weakness. This decompression surgery does not reliably reduce low back pain.

The timing of surgery is multifactorial, based on the duration and severity of symptoms. There is consensus that a cauda equina syndrome (bladder or bowel paralysis) is an absolute indication for immediate surgery. In general, there is a greater than 90% chance of successful resolution of sciatica after surgery.

The surgery can be performed with a microdiscectomy, laminotomy, laminoplasty, or laminectomy technique as indicated by the need for exposure of the affected nerve roots.

In the recent SPORT clinical study (Spine Patient Outcome Research Trial), a randomized, multi-site trial that the HSS Spine Service participated in, patients who underwent surgery for a lumbar disc herniation achieved greater improvement than non-operative treated patients in relief of sciatica, physical function, and bodily pain.

CAM
  • Acupuncture or Acupressure: Practitioners believe your body has an energy force called Qi or Chi (pronounced "chee"). They think that when Chi is blocked, you can develop physical illness. Both acupuncture and acupressure work to restore a healthy, energetic flow of Chi. (These Eastern approaches to healing are different from Western scientific concepts. That doesn't make them better or worse; it just makes them different.)

    In acupuncture, the practitioner inserts fine needles into your body at specific points (it doesn't hurt, honestly). Acupressure is similar to acupuncture— but there are no needles involved. The practitioner uses thumbs, fingers, and elbows instead of needles.

  • Biofeedback: This is more than telling your body, "Stop feeling pain." Biofeedback is a mind-body therapy teaches you how to change, or control a habitual reaction to pain or stress. For example, if it's muscle tension causing your sciatica, you could learn deep-breathing techniques and mental exercises to help you to relax.
  • Yoga: If piriformis syndrome is the cause of your sciatica, yoga can help. Some yoga movements can gently stretch the piriformis muscle. As a word of caution, some yoga positions can make sciatica worse. Avoid positions that involve forward folds, twisting, or stretching the back of your legs. These movements can further irritate your sciatica.

http://www.emaxhealth.com/2/45/33127/sciatica-symptoms-and-treatment.html
http://www.spineuniverse.com/displayarticle.php/article4127.html

Tuesday, September 29, 2009

COMPLICATIONS OF SCIATICA

POSSIBLE COMPLICATIONS OF SCIATICA


a) Partial or complete loss of leg movement
b) Partial or complete loss of sensation in the leg
c) Recurrent or unnoticed injury to the leg
d) Side effects of medications
e) Loss of bowel or bladder function

Partial or complete loss of leg movement


. Partial damage to the nerve may demonstrate weakness of knee flexion (bending), weakness of foot movements, difficulty bending the foot inward (inversion), or bending the foot down (plantar flexion). A person's reflexes may be abnormal, with weak or absent ankle-jerk reflex. Several different tests can be performed to find the cause of sciatic nerve dysfunction.


Partial or complete loss of sensation in the leg
Abnormal sensations such as tingling or numbness which may be due from remaining in the same seated or standing position for a long time, injury to a nerve (eg: a low back injury can cause numbness or tingling down the back of the leg), pressure on the spinal nerves (eg: herniated disc) or pressure on peripheral nerves from enlarged blood vessels, tumors, scar tissue or infection.


Side Effects of Medications
Regular use of NSAIDS may be hazardous as it has been associated with the following side effects:
· Ulcers and gastrointestinal bleeding. This is the major danger with long-term use of NSAIDs. (Indomethacin poses a higher risk than many others for this adverse effect.)
· Increased blood pressure. Most NSAIDs appear to pose this risk, with higher risks observed with piroxicam (Feldene), naproxen (Aleve), and indomethacin (Indocin). (Sulindac has the smallest effect and aspirin as no risk.) People with hypertension, severe vascular disease, kidney, or liver problems and those taking diuretics must be closely monitored if they need to take NSAIDs.
· May delay the emptying of the stomach, which could interfere with the actions of other drugs. The elderly are at special risk.
· Dizziness.
· Tinnitus (ringing in the ear).
· Headache.
· Skin rash.
· Depression has also been noted.
· Confusion or bizarre sensation (in some higher-potency NSAIDs, notably indomethacin).
· Possible higher risk for miscarriage (particularly if the NSAID is taken for more than a week or around the time of conception).
· Kidney abnormalities have been reported in people taking NSAIDs, which resolve when the drugs are withdrawn. Any sudden weight gain or swelling should be reported to a physician. Anyone with kidney disease should avoid these drugs.
· There is a small risk for liver abnormalities.


Loss of Bowel or Bladder Function
Urinary continence, as it is better known involves a strong, sudden need to urinate followed by instant bladder contraction and involuntary loss of urine. You don't have enough time between when you recognize the need to urinate and when you actually do urinate. It could be deemed as sudden or temporary arising from bedrest, during the recovery from surgery.

Causes and Risk Factors of Sciatica

Sciatica refers to pain, weakness, numbness, or tingling in the leg. It is caused by injury to or compression of the sciatic nerve. Sciatica is a symptom of another medical problem, not a medical condition on its own.

Sciatica occurs when there is pressure or damage to the sciatic nerve. This nerve starts in the spine and runs down the back of each leg. This nerve controls the muscles of the back of the knee and lower leg and provides sensation to the back of the thigh, part of the lower leg, and the sole of the foot.

What causes sciatica?
In many cases of sciatica there is no single obvious cause. It is thought that general 'wear and tear' on the body may lead to episodes of sciatica. This is why the condition is more common in people who are over 40 years of age or those who have occupations that involve a lot of physical activity.

Common causes of sciatica include:
• Piriformis syndrome (a pain disorder involving the narrow piriformis muscle in the buttocks)
• Slipped disk
• Degenerative disk disease
• Spinal stenosis
• Pelvic injury or fracture
• Tumors

Herniated disc
A herniated (or slipped) disc is the most common identified cause of sciatica.
Your spine is made up of vertebrae, discs, and nerves. The vertebrae are supported and cushioned by discs of cartilage. As a person gets older the discs start to become harder, tougher and more brittle. Repeated strain on the back means that there is a chance that the hardened discs may split or rupture. If a rupture occurs some of the soft core of tissue inside the disc can press against the sciatic nerve, causing sciatica.

A disk in the lumbar area becomes herniated when it ruptures or thins out, and degenerates to the point that the gel within the disk (the nucleus pulposus) pushes outward. The damaged disk can take on many forms:
•A bulge -- The gel has been pushed out slightly from the disk and is evenly distributed around the circumference.
•Protrusion -- The gel has pushed out slightly and asymmetrically in different places.
•Extrusion -- The gel balloons extensively into the area outside the vertebrae or breaks off from the disk.

Pain in the leg may be worse than the back pain in cases of herniated disks. There is also some debate about how pain develops from a herniated disk and how frequently it causes low back pain. Many people have disks that bulge or protrude and do not suffer back pain. Extrusion (which is less common than the other two conditions) is highly associated with back pain, since the gel is likely to extend out far enough to press against the nerve root, most often the sciatic nerve.

Degeneration
Degeneration is the term often given to the various processes which are normally experienced due to age and use. The spinal structures degenerate just like every other part of our physical bodies. The vast majority of spinal degeneration is completely normal, expected and asymptomatic. Degenerative disc disease is the most common diagnosis, with spinal osteoarthritis not far behind. Specific diagnoses related to osteoarthritis include bone spur formation often blamed for foraminal stenosis and facet joint syndrome. Spinal stenosis is another common process which occurs naturally, mostly due to advanced age. Most of these conditions are not inherently painful and are rarely the source of any significant discomfort.

Spinal stenosis
Spinal stenosis is the narrowing of the spinal canal, or narrowing of the openings (called neural foramina) where spinal nerves leave the spinal column. This condition typically develops as a person ages and the disks become drier and start to shrink. At the same time, the bones and ligaments of the spine swell or grow larger due to arthritis and chronic inflammation. However, other problems, including infection and birth defects, can sometimes cause spinal stenosis. If the narrowing occurs in your lower back, the sciatic nerve may be compressed, leading to sciatica.

Lumbar Degenerative Disk Disease/Spondylosis
Osteoarthritis occurs in joints of the spine, usually as a result of aging, but also in response to previous back injuries, excessive wear and tear, previously herniated discs, prior surgeries, and fractures. Cartilage between the joints of the spine is destroyed and extra bone growth or bone spurs develop. The rate at which these changes develop varies between people..The end result of these changes is a gradual loss of mobility of the spine, narrowing of the spaces for spinal nerves and spinal cord, and drying out or degeneration of the spinal discs. Depending on which part and how much of the spine is involved, symptoms may be similar to that of a herniated disc, lumbar strain, or spinal stenosis (narrowing of the spinal canal).

Most patients will report the presence of gradually worsening history of back pain over time. For others, there may be minimal history of back pain, but at some point in this process any disruption, such as a minor injury that results in disk inflammation, can cause impingement on the nerve root and trigger pain.

Patients may experience pain or numbness, which can occur in both legs, or on just one side. Other symptoms include a feeling of weakness or heaviness in the buttocks or legs. Symptoms are usually present or will worsen only when the person is standing or walking upright. Often the symptoms will ease or disappear when sitting down or leaning forward. These positions may create more space in the spinal canal, thus relieving pressure on the spinal cord or the spinal nerves. Patients with spinal stenosis are not usually able to walk for long periods of time. They may be able to ride an exercise bike.

Spondylolisthesis
Spondylolisthesis is a condition where the discs degenerate to such an extent that they are no longer able to properly support the vertebrae. This may lead to a vertebra slipping forward over the one below it. Spondylolisthesis occurs when one of the lumbar vertebrae slips over another, or over the sacrum. If this occurs in the lower back, the slipped vertebra can compress the sciatic nerve, causing sciatic pain.

In children, spondylolisthesis usually occurs between the fifth bone in the lower back (lumbar vertebra) and the first bone in the sacrum area. It is often due to a birth defect in that area of the spine. In adults, the most common cause is degenerative disease (such as arthritis). The slip usually occurs between the fourth and fifth lumbar vertebrae. It is more common in adults over 65 and women.

Other causes of spondylolisthesis include stress fractures (commonly seen in gymnasts) and traumatic fractures. Spondylolisthesis may occasionally be associated with bone diseases.

Spondylolisthesis may vary from mild to severe. It can produce increased lordosis (swayback), but in later stages may result in kyphosis (roundback) as the upper spine falls off the lower spine.

Symptoms may include:
• Lower back pain
• Pain in the thighs and buttocks
• Stiffness
• Muscle tightness
• Tenderness in the slipped area

Pain generally occurs with activity and is better with rest. Neurological damage (leg weakness or changes in sensation) may result from pressure on nerve roots, and may cause pain radiating down the legs.

Osteoporosis and Compression Fractures
Osteoporosis is a disease of the skeleton in which the amount of calcium present in the bones slowly decreases to the point where the bones become fragile and prone to fractures. It usually does not cause pain unless the vertebrae collapse suddenly, in which case the pain is often severe. More than one vertebra may be affected.

In a compression fracture of the vertebrae, the bone tissue of the vertebra collapses. More than one vertebra may collapse as a result. When the fracture is the result of osteoporosis, the vertebrae in the thoracic (chest) and lower spine are usually affected, and symptoms may be worse with walking.

With multiple fractures, kyphosis (a forward hump-like curvature of the spine) may result. In addition, compression fractures are often responsible for loss of height. Pressure on the spinal cord may also occur, producing symptoms of numbness, tingling, or weakness. Symptoms depend upon the area of the back that is affected; however, most fractures are stable and do not produce neurological symptoms.

Piriformis syndrome
-pressure on the sciatic nerve from the piriformis muscle in the buttocks

The pressure on the sciatic nerve can tighten and irritate the sciatic nerve. Symptoms of piriformis syndrome may include: a sciatica-like pain and/or numbness in the leg that is usually more intense above the knee, usually starts in the rear rather than the low back, and often spares the low back of symptoms or signs.
Piriformis syndrome can mimic the signs and symptoms of sciatica pain from a disc herniation and is part of the different diagnosis of possible causes of sciatica.

Injury
Any significant trauma to the back might damage some of the spinal structures, causing a variety of painful conditions, including sciatica. Vertebral fractures might also influence proper nerve function. While these conditions exist in some patients, they typically either resolve without treatment or will heal with proper medical attention. Injury is seldom a cause of ongoing chronic sciatica.

Ischemia
The most common, yet least diagnosed cause of chronic nerve pain is certainly ischemic sciatica. The harmless, but incredibly painful, oxygen deprivation sciatica process can occur due to disease or injury. However, the majority of patients demonstrate psychosomatic ischemia which is enacted by the subconscious mind. This is the reason why so many sciatic nerve pain syndromes do not respond well to medical or complementary treatment.

Inflammatory Conditions and Arthritis
Inflammatory disorders and arthritis syndromes can produce inflammation in the spine.

Ankylosing spondylitis is a chronic inflammation of the spine that may gradually result in a fusion of vertebrae. Symptoms include a slow development of back discomfort, with pain lasting for more than 3 months. The back is usually stiff in the morning; pain improves with movement or exercise. In severe cases, the patient stands or sits stooped over. It can be quite mild, however, and it rarely affects a person's ability to work. It occurs mostly in young Caucasians in their mid-20s. The disease is more common in men, but about 30% of the cases are in women. Researchers believe that in most cases the cause is hereditary.

http://www.youtube.com/watch?v=MoebKOngjbs



Risk Factors
In most known cases, pain begins with an injury, after lifting a heavy object, or after making a sudden movement. Not all people have back pain after such injuries, however. In the majority of back pain cases, the causes are unknown.

Aging
Intervertebral disks begin deteriorating and growing thinner by age 30. One-third of adults over 20 show signs of herniated disks (although only 3% of these disks cause symptoms). As people continue to age and the disks lose moisture and shrink, the risk for spinal stenosis increases. The incidence of low back pain and sciatica increases in women at the time of menopause as they lose bone density. In older adults, osteoporosis and osteoarthritis are also common. However, the risk for low back pain does not mount steadily with increasing age, which suggests that at a certain point, the conditions causing low back pain plateau.

High-Risk Occupations
Jobs that involve lifting, bending, and twisting into awkward positions, as well as those that cause whole-body vibration (such as long-distance truck driving), place workers at particular risk for low back pain. The longer a person continues such work, the higher their risk. Some workers wear back support belts, but evidence strongly suggests that they are useful only for people who currently have low back pain. The belts offer little added support for the back and do not prevent back injuries.

A number of companies are developing programs to protect against back injuries. However, studies have been mixed on the outcome of company interventions. Employers and workers should make every effort to create a safe working environment. Office workers should have chairs, desks, and equipment that support the back or help maintain good posture.

Low back pain accounts for significant losses in workdays and dollars. According to the Bureau of Labor Statistics, back pain was responsible for around 60% of cases of people missing work due to pain involving the upper body. A 2004 study analyzed health care expenses in the United States. The analysis found back pain cost over $90billion, of which $26 billion was spent directly on treating the back pain.

Osteoporosis is a condition characterized by progressive loss of bone density, thinning of bone tissue, and increased vulnerability to fractures. Osteoporosis may result from disease, dietary or hormonal deficiency, or advanced age. Regular exercise and vitamin and mineral supplements can reduce and even reverse loss of bone density.



Medical Conditions in Children
Persistent low back pain in children is more likely to have a serious cause that requires treatment than back pain in adults.

Stress fractures (spondylolysis) in the spine are a common cause of back pain in young athletes. Sometimes a fracture may not show up for a week or two after an injury. Spondylolysis can cause spondylolisthesis, a condition in which the spine becomes unstable and the vertebrae slip over each other.

Hyperlordosis is an inborn exaggerated inward curve in the lumbar area. Scoliosis, an abnormal curvature of the spine in children, does not usually cause back pain.

Juvenile chronic arthropathy is an inherited form of arthritis. It can cause pain in the sacrum and hip joints of children and young people. It used to be grouped under juvenile rheumatoid arthritis, but is now defined as a separate problem.

Injuries can also cause back pain in children.

Pregnancy
Pregnant women are prone to back pain due to a shifting of abdominal organs, the forward redistribution of body weight, and the loosening of ligaments in the pelvic area as the body prepares for delivery. Tall women are at higher risk than short women.

Psychological and Social Factors
Psychological factors are known to play a strong influential role in three phases of low back pain:

•Some evidence suggests preexisting depression and the inability to cope may be more likely to predict the onset of pain than physical problems. A "passive" coping style (not wanting to confront problems) was strongly associated with the risk of developing disabling neck or low back pain.

•Social and psychological factors, as well as job satisfaction, all play a role in the severity of a person's perception of back pain. For example, one study compared truck drivers and bus drivers. Nearly all the truck drivers liked their work. Half of them reported low back pain but only 24% lost time at work. Bus drivers, on the other hand, reported much lower job satisfaction than truck drivers, and these workers with back pain had a significantly higher absentee rate than truck drivers in spite of less stress on their backs. Similarly, another study found that pilots, who generally reported "loving their jobs," reported far fewer back problems than their flight crews. And yet another study reported that low rank, low social support, and high stress in soldiers was associated with a higher risk for disabling back pain.

•Depression and a tendency to develop physical complaints in response to stress also increase the likelihood that acute back pain will become a chronic condition. The way a patient perceives and copes with pain at the beginning of an acute attack may actually condition the patient to either recover or develop a chronic condition. Those who over-respond to pain and fear for their long-term outlook tend to feel out of control and become discouraged, increasing their risk for long-term problems.

Studies also suggest that patients who reported prolonged emotional distress have less favorable outcomes after back surgeries. It should be strongly noted that the presence of psychological factors in no way diminishes the reality of the pain and its disabling effects. Recognizing this presence as a strong player in many cases of low back pain, however, can help determine the full range of treatment options.

Diagnosis of Radiculopathy

I took all my info from this website....

http://emedicine.medscape.com/article/95025-diagnosis

If you need the powerpoint presentation, come get it from me on friday (:

Neurologic Examination Resource

Disclaimer: I didn't read any of these (yet), but it seemed like a good resource so I'm just going to put it up. If its trash, feel free to relentlessly attack me with asparagus stalks and reduce this post to cyber dust.

Also, just a minor note, I have no idea why I could access all these, since I get prompted for a login when I attempted to track the root webpage. I only got chapter 1 off Google - the rest was by manipulating the address. /shrug


Oh, and some chapters have several sections, in case you didn't notice. :)

http://www.aan.com/go/education/curricula/family/chapter1/section1
The basics - neurological history, systems review & neuromuscular examinations.

http://www.aan.com/go/education/curricula/family/chapter2/section1
On visual problems.

http://www.aan.com/go/education/curricula/family/chapter3/section1
On numbness.

http://www.aan.com/go/education/curricula/family/chapter4/section1
On weakness.

http://www.aan.com/go/education/curricula/family/chapter5/section1
On dizziness.

http://www.aan.com/go/education/curricula/family/chapter6/section1
On headaches.

http://www.aan.com/go/education/curricula/family/chapter7/section1
On episodic disorders. [The seizure/epilepsy family]

http://www.aan.com/go/education/curricula/family/chapter8/section1
On gait & movement disorders.

http://www.aan.com/go/education/curricula/family/chapter9/section1
On neck & back pain.

http://www.aan.com/go/education/curricula/family/chapter10/section1
On common neurologic emergencies.

http://www.aan.com/go/education/curricula/family/chapter11/section1
On changes in behavior.

http://www.aan.com/go/education/curricula/family/chapter12/section1
On sleep disorders.

http://www.aan.com/go/education/curricula/family/chapter13/section1
On common problems in pediatric neurology.

That's it.

Monday, September 28, 2009

General Overview of Management of Sciatica & Underlying Causes.

Sciatica
By JAMA.
Diagnosis
» Complete history & physical examination to locate irritated nerve root.
» X-rays to detect spondylolisthesis (vertebral misalignment), narrowed discs, or erosion suggesting spinal tumors; cannot detect herniated disc.
» MRI allows visualization of IV discs, ligaments, muscles, & tumors.
» CT myelography using contrast dyle injected into the spine allows visualization of the spinal cord & nerves.

Treatment
» Analgesics (NSAIDs, steroidal drugs/injection).
» Physical therapy.
» Surgery if all else fails, if symptoms have progressed, or if bladder/bowel incontinence occurs.

Sciatica
By Mayoclinic.
Diagnosis & Investigation
» History.
» Physical exam.
» Spinal X-ray.
» MRI.
» CT scan.

Complications
» Potentially cause permanent nerve damage
»»» Loss of feeling
»»» Loss of movement
»»» Loss of bowel/bladder function

Treatment & Drugs
» Self-care measures proven effective.
» Physical therapy.
» Prescription drugs
»»» NSAIDs
»»» Muscle relaxant
»»» Narcotics
»»» Tricyclic antidepressants
»»» Anticonvulsants

The Big Guns
» Epidural steroid injections. (corticosteroids)
» Surgery. (lumbar laminectomy, discectomy, microdiscectomy)

Prevention
» Regular exercise.
» Proper sitting posture.
» Good body mechanics.

Lifestyle & Home Remedies
» Avoid aggravating activities.
» Cold packs.
» Hot packs.
» Stretching.
» Over-the-counter drugs. (analgesics)
» Regular exercise.

CAM
» Acupuncture.
» Chiropractic.
» Massage.
» Hypnosis.

IV Disc Herniation
By AANS.
Lumber Spine Herniation
» Sciatica frequently results.
» Pressure on the nerves preceding the sciatic nerves causes pain, burning, tingling, & numbness that radiates from the buttock into the leg & sometimes the foot.
» Usually unilateral.
» Pain described as sharp & electric shock-like
» May be aggravated on standing/walking/sitting.
» Associated with low back pain.

Cervical Spine Herniation
» Sharp pain in the neck/between the shoulder blades, radiating down the arm to the hand/fingers.
» Numbness & tingling in the shoulder/arm.
» May be aggravated by certain positions/neck movements.

Diagnosis
» History.
» Symptoms.
» Physical examination.
» X-ray
»»» Shows vertebrae structure & joints outline; obtain to search for other potential causes of pain (tumour, infection, fracture etc.)
» CT/CAT scan
»»» Shows shape & size of spinal canal, its contents, & surrounding structures.
» MRI
»»» Shows spinal cord, nerve roots, & surrounding strucutres; also enlargement, degeneration, & tumours.
» Myelogram
»»» Shows pressure on spinal cord/nerves due to herniated discs/bone spures/tumours.
» Electromyogram & Nerve Conduction Studies
»»» Indicate ongoing nerve damage/healing nerve from past injury/nerve compression.

Treatment
» Surgery not usually required.
» Initial treatment conservative & non-surgical
»»» Bed rest
»»» Maintain period of low, painless activity level
»»» NSAIDs
»»» Epidural steroidal injection
»»» Physical therapy (pelvic traction, gentle massage, ice & heat therapy, ultrasound, electrical muscle stimuatlion, & stretching exercise)

Surgery
» Alternative if conservative methods fail
» Considerations for candidacy
»»» Back & leg pains limits normal activity/impairs quality of life
»»» Development of progressive neurological deficits
»»» Bowel & bladder incontinence
»»» Difficulty standing/walking
»»» Medication & physical therapy ineffective
»»» Patient in reasonably good health
» Surgical options
»»» Artificial disc surgery - surgical replacement of the injured lumbar disc with a manufactured one
»»» Discectomy - complete/partial surgical removal of an IV disc
»»» Laminectomy - surgical removal of most of the lamina of a vertebra
»»» Laminotomy - an opening made in a lamina to relieve pressure on nerve roots
»»» Spinal fusion - bone is grafted onto the spine, creating a solid union between two or more vertebrae.

Lumar Spine Surgery
» Lumbar laminotomy
»»» Incision down the center of the back over site of herniated disc
»»» Muscles moved to expose vertebrae
»»» Small opening made between the 2 vertebrae to gain access to gain access to herniated disc
»»» Disc is removed via disecectomy
»»» Spine is stabilized, often via spinal fusion
» Lumbar laminectomy
»»» For more involved cases
» Artificial disc surgery
»»» Incision made through abdomen
»»» Damaged disc removed & replaced
»»» Strict conditions for candidacy
»»»»» Must have disc degeneration in only 1 disc, between L4 & L5;
»»»»» Must have undergone at least 6 months of treatment without signs of improvement
»»»»» Must be in overall good health without infection/osteoporosis/arthritis

Cervical Spine Surgery
» Surgery about the neck (posterior/anterior) depends on the exact location of the herniated disc & the experience & preference of the surgeron.
» Laminotomy followed by discectomy , subsequently spine stabilization.

Postsurgery
» Specific instructions on physical activity & pain medication prescribed.

Piriformis Syndrome
By NNDS.
The Nut Shell
» Rare neuromuscular disorder.
» Piriformis muscle compresses/irritates the sciatic nerve.
» Pain is aggravated by prolonged sitting/climbing stairs/walking/running.

Treatment
» Stretching exercises.
» Massages.
» Anti-inflammatory drugs.
» Cessation of aggravating activities.
» Corticosteroid injection at junction of piriformis-sciatic nerve.
» Surgery.

Prognosis
» Two thumbs up.

Anatomy Dissections

Remember those dissection videos I showed before the Raya holidays - Clickie.

There's also another page on neuroscience with some neuroanatomy - Clickie.

Neuroscience dissection is here - Clickie.

You can save the videos if you want to, but it involves a bit of work - which I'm too lazy to type out here. You can either Google on how to save Quicktime files, or just bug me about it irl (in real life).

Sciatica Symptoms

The most common symptom from sciatica is PAIN. Most people describe a deep, severe pain that starts low on one side of the back and then shoots down the buttock and the leg with certain movements.

The pain from sciatica is felt along the sciatic nerve and can be felt deep in the buttock, with pain that travels down the back of the leg, sometimes to the foot. The pain can be accompanied by tingling, ‘pins and needles’, or numbness, and sometimes by muscular weakness in the leg.

Sciatica is usually felt in only one leg at a time. Sometimes, a sensation like an electric shock can be felt along the nerve. The pain can range from a mild ache to incapacitating pain.

· The pain is usually worse with both prolonged sitting and standing. Some people describe the worst pain when trying to stand from a low sitting position, such as standing up after sitting on a toilet seat.

· In most people, the pain is made worse by sneezing, coughing, laughing, or a hard bowel movement. Bending backward can also make the pain worse.

· You may also notice a weakness in your leg or foot, along with the pain. The weakness may become so bad you can't move your foot.

Most sciatica gets better within a few weeks and doesn’t result in permanent damage. If your pain doesn’t go away after a couple of weeks, see your doctor. If you lose control of your bladder or bowels or you have severe pain, weakness of your muscles, or numbness, seek medical attention immediately.

Different Types of Sciatica Pain

· Sciatica from L4 nerve root (usually the L3-L4 level)
The patient may have reduced knee-jerk reflex. Symptoms of sciatica stemming from this level of the lower back may include: pain and/or numbness to the medial lower leg and foot; weakness may include the inability to bring the foot upwards (heel walk).

· Sciatica from L5 nerve root (usually the L4-L5 level)
The patient may have weakness in extension of the big toe and potentially in the ankle (called foot drop). Symptoms of sciatica originating at this level of the lower back may include: top of the foot pain and/or numbness, particularly in the web between the great toe (big toe) and the second toe.

· Sciatica from S1 nerve root (the L5-S1 level)
The patient may have reduced ankle-jerk reflex. Symptoms of sciatica originating at this level of the spine may include: pain and/or numbness to the lateral or outer foot; weakness that results in difficulty raising the heel off the ground or walking on the tiptoes.

· Pressure on the sacral nerve roots from sacroiliac joint dysfunction
Symptoms may include: a sciatica-like pain or numbness that is often described as a deep ache, inside the leg more so than a linear, well-defined geographic area of pain/numbness found in true sciatica.

· Pressure on the sciatic nerve from the piriformis muscle
The pressure on the sciatic nerve can tighten and irritate the sciatic nerve (called piriformis syndrome). Symptoms of piriformis syndrome may include: a sciatica-like pain and/or numbness in the leg that is usually more intense above the knee, usually starts in the rear rather than the low back, and often spares the low back of symptoms or signs.

Piriformis syndrome can mimic the signs and symptoms of sciatica pain from a disc herniation and is part of the different diagnosis of possible causes of sciatica.

VIDEO: http://www.spine-health.com/video/sciatica-interactive-video

Friday, September 25, 2009

is it the SKY ATTIC AH?

Haha! not really sure what this weeks PCL is about but im guessing its sciatica (hence sky-attic-ah)...

Here's what we can collect from the passage:

Name: Adnan Huskic
Age: 43 years old
Occupation: Currently working as a self-employed taxi driver. Use to work as a veterinarian and cattle treater. (Highlights occupation as one of the risk factors for musculoskeletal and nerve problems)

HPI.
When: Has history since 10 years ago and recently on one Saturday night which lasted till the following Monday. Discomfort increased within 48 hours of occurrence of pain.
Where: Pain is his lower back and right leg.
Quality: Sharper than usual. Sever and persistent(48 hours later)
Quantity: No indication but however has gotten worse within 48 hours of occurrence of pain.
Alleviating:
He thought that warm things eg. hot drink, hot water bottle, warm bed and walking occasionally would ease the pain but it DID NOT EASE IT.
Aggravating: Long periods of sitting, lifting heavy suitcases.
Associated Symptoms: Pain spread from the leg to back of right leg into his foot.
+
Beliefs: No beliefs were apparent from the passage.

PAST MEDICAL HX.
Past Medical Hx: Nagging lower back pain since ten years ago. Apart from that seems healthy.
Past Surgical Hx: -
Hospitalizations: -

FAMILY, DRUGS, ALCOHOL, SEXUAL HXx - Not Known.

Social HX.
Occupation: As mentioned earlier, he works as a taxi driver. He works for 60 long and stressful hours a week. ): Both his past and present jobs required him to move heavy objects.
Travel: Because of job, he travels here and there in taxi. Latest to Taiping. Out of country known although he has been in Bosnia.
Migration: Moved from Bosnia 8 years ago.
Family: Has 3 children with wife Ziba, that works as a cleaner in private hospital.
Financial: Hard to make ends meet. Wife does not make much either.

Discussion Points.
Sociological Issues:
a) The occupation as a risk factor ie. involved in pathogenesis of medical condition.
b) Stresses of migrating and settling down in a new country.
c) Financial burdens as one of the stressors faced by people. Ie. Adnan felt worried bout it affecting his work.
d) Financial help or assistance for these people?

Others:
a) Limit of working hours for normal working people?
b) Normal remedies for treating backaches.

Differential Diagnosis:
a) Osteoarthritis - Bony spurs causing compression of nerves
b) Herniation of Intervetebral Disc
c) Torn ligament due to hyperflexion or hyperextension
d) Muscle tear or pulled muscle
e) Sciatica
f) Piriformis Syndrome
g) Osteoporosis
h) Tumor
i) Trauma
j) Fracture of the bone (spine) and compression fracture (JUST learned that from Grey's Anatomy)
k) Osteomyelitis
l) Vascular complications
m) Mutiple Sclerosis
n) Avascular necrosis bone
o) Ischemic conditions causing necrosis of muscle
p)Cauda Equina Syndrome
q) Spondylolisthesis

All these were the diagnosis that I could come of with. Note that most of this can actually lead to sciatica. However, in diagnosis a patient, sciatica might be a complication that arises from an underlying medical condition. So in this case, it is wise to explore all the possible options including those that are involved in the aetiology of sciatica.

Now that I think that the problem might be sciatica, here's some sources about it.


Since we need a authoritative, accurate, current and objective source, here's once from JAMA.
A little simple explanation from them about sciatica (:


There's also a specific syndromes called the piriformis syndrome and the cauda equina syndrome. Basically also sciatica but sciatica can be due to a host of reasons.


Whether or not this is caused by a host of different reasons, it is safe to say that it is most likely sciatica, which is a condition for compressed sciatic nerve. This few correlations tell why:-

Why this may be a neurological condition, the very mere fact that the pain goes from the back to his leg and radiated to his foot indicates that the pain is caused by a part of the body that is connected to all these regions. According to as much as i know, I don't think any muscles stretch that long all the way down to the foot. Only nerves and blood vessels has this connection. Given that we are dealing with pain, it is most likely involving the nerves being irritated and compressed. Since the pain is not superficial, we can assume this involves direct pain on the nerve which are deep and not the superficial eg subcutaneous ones.

Pain on lower back which radiates all the way down to his foot. The sciatic nerve is one of the major nerve that supplies the lower limbs. Most important and most likely to be compressed.

The pain is only on one of his leg. Sciatica may usually occur sometimes as a unilateral pain.

Adnan is in a suitable age at risk for sciatica. Age risk is from 30-50 years old. This also spells the risk for possible degenerative arthritis which may result in sciatica.

Given that Adnan lifts heavy things, he has the tendency to cause a ruptured ligament or IV disc. This also points towards sciatica.

Given that the pain got worse and that normal remedies such as rest and hot water bottles do not work, it may seem like the pain is not caused superficially at the muscles but in the deeper regions involving the spine, nerves or ligaments.


SO YEAH, thats my two cents for this coming weeks PCL. Remember to READ TALLY O' CONNOR for DR. ARUL'S session and DO ANATOMY FOR THIS COMING WEEK!!!!

Enjoy the holidays, or what's left of it (:

Friday, September 18, 2009

Summarized Epidemiology

Self-reported prevalence

In the 2001 NHS, about 75 out of 1,000 Australians reported osteoarthritis. This equates to around
1.4 million people. This estimate is based on the NHS question: whether the survey respondent ‘currently has osteoarthritis’.

The NHS survey assumes all reported cases of osteoarthritis to be long term (i.e. conditions that
have lasted at least six months, or that are likely to last six months or more). The prevalence of osteoarthritis increases with age: relatively few people at younger ages report having it. By age 55, however, the prevalence rises sharply (ABS 2002).

Osteoarthritis is reported more frequently by females than males (92 compared with 57 per 1,000 persons in 2001). The difference persists across all ages.
In 2001,
• the prevalence was 331 per 1,000, among females aged 65–74.
• 374 per 1,000 among those aged 75 and over.
• Comparable rates among males that year were 186 and 236 per 1,000, respectively


• Several studies have reported a crossover in osteoarthritis prevalence between the two sexes around the age of 45.
• Males are affected more commonly below age 45, whereas above age 45 females are affected not only more frequently but also more severely .

• In an omnibus survey of the South Australian population, Hill et al. (1999) estimated the prevalence of osteoarthritis among those aged 15 and above to be around 86 per 1,000 persons (51 per 1,000 males and 111 per 1,000 females). The prevalence increased with age, rising above 261 per 1,000 among those aged
70 and above.
• A study in North Sydney estimated the prevalence of osteoarthritis to be around 79 per 1,000 persons

• Symptomatic osteoarthritis was also reported by more than one-quarter of persons aged 60 and above in the Dubbo Osteoporosis Study (Jones et al. 1995).

• The NHS indicates that the prevalence of osteoarthritis has risen from 69 per 1,000 persons in 1995 to 75 per 1,000 persons in 2001.

Thursday, September 17, 2009

Prevalence and Incidence

Prevalence and incidence
The proportion of people in the general population who experience osteoarthritis is a useful measure of its
impact. For an intermittent episodic problem such as osteoarthritis, prevalence needs to be measured across
a defined period of time. Regular national data, based on self-reports, are now available about its prevalence
through the National Health Surveys conducted by the Australian Bureau of Statistics. However, no national data
based on case definition by physical examination or radiological evidence are available.
The incidence of osteoarthritis can be modelled using the prevalence data and other epidemiological parameters
(AIHW: Mathers & Penm 1999). There are no direct sources for this information.
Self-reported prevalence
In the 2001 NHS, about 75 out of 1,000 Australians reported osteoarthritis. This equates to around
1.4 million people. This estimate is based on the NHS question: whether the survey respondent ‘currently has
osteoarthritis’. The NHS survey assumes all reported cases of osteoarthritis to be long term (i.e. conditions that
have lasted at least six months, or that are likely to last six months or more). The prevalence of osteoarthritis
increases with age: relatively few people at younger ages report having it. By age 55, however, the prevalence
rises sharply (ABS 2002).
Osteoarthritis is reported more frequently by females than males (92 compared with 57 per 1,000 persons in
2001). The difference persists across all ages. In 2001, the prevalence was 331 per 1,000, among females
aged 65–74, rising to 374 per 1,000 among those aged 75 and over. Comparable rates among males that year
were 186 and 236 per 1,000, respectively (Figure 3.2).
Several studies have reported a crossover in osteoarthritis prevalence between the two sexes around the
age of 45. Males are affected more commonly below age 45, whereas above age 45 females are affected not
only more frequently but also more severely (Kelsey & Hochberg 1988). No such crossover was noted in the
NHS self-reports.
Other regional/jurisdictional sources confirm the high prevalence of osteoarthritis in Australia.
• In an omnibus survey of the South Australian population, Hill et al. (1999) estimated the prevalence of
osteoarthritis among those aged 15 and above to be around 86 per 1,000 persons (51 per 1,000 males and
111 per 1,000 females). The prevalence increased with age, rising above 261 per 1,000 among those aged
70 and above.
• A study in North Sydney estimated the prevalence of osteoarthritis to be around 79 per 1,000 persons
(March et al. 1998).
• Symptomatic osteoarthritis was also reported by more than one-quarter of persons aged 60 and above in the Dubbo Osteoporosis Study (Jones et al. 1995).
There is no regular time series available on the prevalence of osteoarthritis in Australia. The NHS indicates
that the prevalence of osteoarthritis has risen from 69 per 1,000 persons in 1995 to 75 per 1,000 persons in
2001. The Survey of Disability, Ageing and Carers (SDAC) categorises osteoarthritis together with other forms of
arthritis. No comparative information on that account is therefore available.
Radiological evidence
The radiological evidence suggests much higher prevalence of osteoarthritis than the self-reports. Osteoarthritisrelated
changes were noted on x-ray in more than 50% of persons over the age of 65, and almost universally in
those after age 85 in North Sydney (March 1997).
The radiographic evidence is based on the presence of osteophytes, joint space narrowing, subchondral cysts
and bone remodelling, with the severity of the condition, graded from none (0) through doubtful (1), minimal (2)
and moderate (3) to severe (4). One of the problems with this case definition is that many people with positive
x-ray findings report no pain or disability (Lawrence et al. 1966). Conversely, some individuals report pain but
show no radiological evidence. In addition, primary sources of data on osteoarthritis are based on radiographs of
only a few joints in each person (McDuffie et al. 1987).
Incidence
Direct estimation of the incidence of osteoarthritis is difficult. The Australian Burden of Disease Study, using
DISMOD software to model epidemiological parameters, estimated the incidence of radiological osteoarthritis in
Australia to be 2.9 per 1,000 females and 1.7 per 1,000 males (AIHW: Mathers & Penm 1999). This translates
to some 27,000 new cases annually. The incidence increases with age. It is highest among females between the
ages of 65 and 74 (14 per 1,000) and among males aged 75 and over (9 per 1,000).
To date, no prospective population-based study has been undertaken in Australia to estimate the incidence of
osteoarthritis. More recent longitudinal surveys in the United Kingdom suggest that the incidence may be higher,
with 20–30 per 1,000 females aged 50 to 60 developing new radiological knee, hip and spinal osteoarthritis
each year.
Estimating the prevalence and incidence of osteoarthritis is complicated by a variety of factors. The estimates
may vary depending upon the number of joints studied, the age and sex of the respondents, and the reporting
method used (physical examination, x-ray, self-report). The correspondence between the radiological evidence,
clinical features and self-assessment is also variable.

Tuesday, September 15, 2009

Management and Prevention of Osteoarthritis

MANAGEMENT OF OSTEOARTHRITIS
Non-pharmacological management

Education and behavioural intervention
Education of patients with OA can reduce their pain and improve their quality of life.The aim is to provide patients with an understanding of the disease process, its prognosis and the rationale and implications of managing their condition. Patients can be educated during the consultation with a doctor, through consumer groups or by being provided with written material.

Weight Loss
Obesity is a risk factor for the development of OA, and is associated with radiological progression of the disease, and disability.When people walk, three to six times their body weight is transferred across the knee joint; any excess weight should be multiplied by this factor to estimate the excess force across the knee joint of overweight people.Small studies of overweight patients with knee OA have shown that modest weight loss (< 5 kg) has significant short-term and long-term reduction in symptoms of OA.
In managing OA, weight reduction should be a key goal. Exercise plays a role, but pain and disability can make it difficult for patients to exercise sufficiently to lose weight. Weight loss can be achieved with regular sessions with a dietitian who can provide instruction on reducing caloric intake and the use of food diaries, and cognitive-behavioural modification to change dietary habits.

Exercise
The aim of exercise is to reduce pain and disability by strengthening muscle, improving joint stability, increasing the range of movement and improving aerobic fitness. Other, theoretical benefits include better self-esteem, weight reduction and improved general health.
Systematic reviews of short-term exercise programs show a small to moderate reduction of pain and disability, with similar benefits seen regardless of the type of exercise.Many such programs have involved intensive supervision and sophisticated equipment, with their sustainability over time unknown. A simple, largely unsupervised, home-based exercise program has been shown to reduce knee pain and disability, with its effects sustained over two years.Although hydrotherapy is widely used, no trials have shown any advantage over land-based exercise. Anecdotally, patients enjoy hydrotherapy and it may be a gentle start in encouraging exercise.
Osteoarthritis at different sites requires different approaches. Range-of-motion exercises may exacerbate pain in OA of the hip, and extension exercises can worsen the pain in patellofemoral OA. Thus, an individual exercise program should be formulated with the patient in consultation with a doctor, physiotherapist or qualified fitness instructor.

Mechanical aids
Although there is no evidence available from well-designed trials to support the efficacy of walking sticks in OA, they are widely recommended.Patients should be encouraged to wear shock-absorbing footwear with good mediolateral support, adequate arch support and calcaneal cushion. Short-term studies have shown biomechanical aids are effective in reducing OA-related pain. Lateral heel wedges may reduce pain related to OA of the medial tibiofemoral compartment,and applying adhesive tape to the patella can provide relief in patellofemoral OA.In patients with significant varus deformity, use of a tube-like knee support made of neoprene or an unloader brace both reduced pain over 6 months, with the brace being slightly more effective.Both may be uncomfortable to wear and the brace is expensive. Physiotherapy and occupational therapy assessment are recommended if there is functional limitation secondary to OA.


Pharmacological management

Pharmacological management should be considered an adjunct to non-pharmacological measures. Drug therapy should be individualised after a careful assessment of symptom severity, comorbid conditions, concomitant therapy, side effects, cost of therapy and patient preferences.

Systemic drugs
Paracetamol
It is widely accepted that paracetamol is the oral analgesic of first choice and, if successful, should be taken long term.Although some patients prefer non-steroidal anti-inflammatory drugs (NSAIDs), paracetamol should be used as initial therapy based on relative cost and safety. It should be taken in divided doses, at regular intervals, with the total daily dose not exceeding 4 g. Although it is one of the safest analgesics, paracetamol can be associated with clinically important adverse events. Paracetamol may prolong the half-life of warfarin, so patients taking warfarin must have their INR (international normalised ratio) monitored closely and their warfarin dose adjusted if necessary.Paracetamol should be used with caution in patients who have liver disease and those with a history of excessive alcohol consumption.

NSAIDs and cyclo-oxygenase-2-specific inhibitors
NSAIDs should be considered only for patients who do not obtain adequate pain relief with paracetamol. Clinicians now have a choice between conventional NSAIDs and cyclo-oxygenase-2-specific (COX-2) inhibitors. COX-2 inhibitors have similar analgesic effects to those of non-selective NSAIDs, with a gastrointestinal (GI) side-effect profile and incidence of endoscopic ulceration similar to placebo.Both NSAIDs and COX-2 inhibitors may cause acute deterioration in renal function, fluid retention and hypertension. The newer COX-2 inhibitors are considerably more expensive than NSAIDs, and uncertainty remains about potential increased risk of cardiovascular events. Cardiovascular risk factors should be considered and patients should be counselled before prescribing rofecoxib (Box 4).

The choice between NSAIDs and COX-2 inhibitors should be made after carefully assessing the risk of serious upper-GI complications and discussing with patients the risk of serious thrombotic cardiovascular events. Patients with no risk factors should use conventional NSAIDs, commencing at a low dose, with dose titration against effect. NSAIDs should be used on an as-required basis, although this often means continuous use. All NSAIDs have similar efficacy, so those with the lowest risk profile for upper-GI haemorrhage (ibuprofen and diclofenac) are recommended. COX-2 inhibitors are recommended for patients with any GI risk factors.Rofecoxib should be avoided in patients with known risk factors for cardiovascular disease. All patients prescribed NSAIDs and COX-2 inhibitors should be counselled about the symptoms of upper-GI haemorrhage and monitored for new or severe upper-GI symptoms. NSAIDs should never be used in combination, except with low-dose aspirin for cardioprotection.
For patients with any risk factors for deterioration in renal function, NSAIDs and COX-2 inhibitors should only be prescribed after very careful consideration of all other options. Plasma sodium, potassium and creatinine levels, blood pressure and the presence of oedema should be checked at baseline and regular intervals.

Glucosamine and chondroitin
Glucosamine sulfate (GS) and chondroitin sulfate (CS) are derivatives of glycosaminoglycans found in articular cartilage, and are available without prescription from pharmacies and supermarkets.Oral GS is able to reduce pain from
20%-25% in patients with mild to moderate primary knee OA.GS is contraindicated in seafood allergy, but is otherwise well tolerated and causes no major side effects. GS should be used at a dose of 1500 mg per day as a divided dose for at least 3 months to determine whether it is therapeutic in any given patient. Topical application of GS and CS may be effective in reducing pain from knee OA.

Opioids
The combination of codeine and paracetamol provides better analgesia than paracetamol alone. However, nausea, vomiting, dizziness and constipation lead to discontinuation of this combination in up to a third of patients.Some patients with intractable pain, who are unsuitable for arthroplasty, may require stronger opiate analgesia. Tramadol is a centrally acting synthetic opioid which inhibits the reuptake of serotonin and noradrenaline. It is generally well tolerated, but is contraindicated in seizure disorders, as it lowers the seizure threshold, and in combination with selective serotonin reuptake inhibitors because of the risk of serotoninergic syndrome.


Topical analgesia
Topical treatment is appropriate for patients as an adjunct to simple analgesia, monotherapy for a single symptomatic joint, or for patients who cannot tolerate systemic therapy. Topical capsaicin has a modest analgesic effect.A local burning sensation is common, but decreases with continued use. Patients must avoid inadvertently transferring the capsaicin to eyes or mucous membranes.

For optimal results, management of OA requires multiple therapies and an individualised approach. Patients need to be involved in formulating and executing the management plan. As the disease progresses, or as comorbid conditions develop, management may need to be revised. The doctor’s role is to coordinate non-pharmacological approaches, supervise pharmacological management with the aim of minimising toxicity, and empower patients to manage their chronic condition.


PREVENTION OF OSTEOARTHRITIS
Weight control. Maintaining a healthy weight may be the single most important thing you can do to prevent osteoarthritis.Being overweight puts extra strain on the joints, particularly the large weight-bearing joints such as the knees, the hips, and the balls of the feet. It is estimated that every 1lb of body weight means at least 3lb of stress at the knee joint, and even more at the hip joint. That would mean that losing just 5lb would take at least 15lb of stress off your knees. Extra weight may also alter the normal structure of the joint and increase the risk for osteoarthritis. Maintain a healthy weight to prevent or reduce joint damage and lower the stress on osteoarthritic joints.

Injury prevention. Protect your joints from serious injury or repeated minor injuries to decrease your risk of damaging cartilage. Repeated minor injuries include those from job-related activities such as frequent or constant kneeling, squatting, or other postures that place stress on the knee joint.

Exercise. Exercise can help reduce joint pain and stiffness. Light- to moderate-intensity physical activity may prevent a decline in, and may even restore, health and function.But some people with osteoarthritis may be reluctant to exercise because of joint pain after activity. You can take various steps to help relieve pain, such as heat and cold therapy or taking pain relievers, which may make it easier for you to exercise and stay active. Choose partial– or non–weight-bearing exercise, such as bicycling, swimming, or water exercise. You can also try light weight-lifting exercises, with supervision.

Research shows that even modest weight loss combined with exercise is more effective in decreasing pain and restoring function than either weight loss or exercise alone.

Young adults who have significant knee injuries have an increased risk of future osteoarthritis. Prevention of joint injuries during youth depends in good part on the use of proper sports equipment and on playing under safe playing conditions. A young person who has a serious knee injury can limit further damage by using a brace to stabilize the knee joint and by changing the way he or she does high-impact exercise.

Heat and cold therapy for osteoarthritis
For moderate to severe pain from osteoarthritis, try applying heat and cold to the affected joints as appropriate. Experiment with these heat and cold techniques until you find what helps you most.
-Apply heat 2 or 3 times a day for 20 to 30 minutes, using a heating pad, hot shower, or hot pack. Heat seems to be effective for pain and stiffness related to inactivity of a joint.
-Try putting cold packs on a painful joint for 10 to 20 minutes. Do not apply a cold pack directly to bare skin. Put a thin towel or pillowcase between the ice and your skin.
-Try ice massage. A small study showed that ice massage for 20 minutes, 5 days per week, improved range of motion and function, although its effect on pain was less clear.
-Try alternating between heat and cold.
-After a heat or cold treatment, try some gentle massage for relaxation and pain relief.
-Paraffin wax is a form of moist heat that may help if you have pain and stiffness in your hands or feet. It is especially useful before exercise. Your physical therapist can teach you to use paraffin at home.

Monday, September 14, 2009

Superficial Anatomy of Buttock

Activity 1: Bones
1.1
Ilium, ischium, pubis
1.1.1
Ischium
1.1.1.1
Posterior division of anterior rami.

1.2


1.2.1.1
Bone marrow harvesting
1.2.2.1
Tensor of fascia latae, sartorius
1.2.3.1
A contusion (bruise) on the pelvis caused by a direct blow to an iliac crest. It usually causes bleeding into the hip abductor muscles, which move legs sideways, away from the midline of the body. This bleeding into muscle tissue creates swelling and makes leg movement painful. The injury usually last from one to six weeks, depending on the damage.

1.2.7.1
Superior gemelli
1.2.8.1
Sacrotuberous ligament, sacrospinous ligament and ischial spine. The sacrotuberous ligament extends across the sciatic notch converting the notch into a foramen that is further subdivided by the sacrospinous ligament and ischial spine into the greater and lesser sciatic foramen.
1.2.9.1
It is the site where the body’s weight rests on.
1.2.9.2
Inferior part of buttock when thigh is flexed.
1.2.9.3
The sciatic nerve extends from a point midway between the greater trochanter and the ischial tuberosity down the middle of the posterior aspect of the thigh. Hamstrings, biceps femoris and semitendinosus which attach proximally to the ischial tuberosity may avulse.

1.2.12.1
Inguinal ligament and indirect muscle attachments.
1.2.12.2
Provides the landmark for palpation of femoral pulse. By placing the tip of the little finger (of the right hand when dealing with the right side) on the ASIS and the tip of the thumb on the pubic tubercle, the femoral pulse can be palpated with the midpalm just inferior to the midpoint of the inguinal ligament by pressing firmly.

1.3.1
Obturator nerve and obturator artery.
1.3.2
Inferior epigastric artery.
1.3.2.1
Could be involved in strangulated femoral hernia. Surgeons placing staples during endoscopic repair of both inguinal and femoral hernias must be careful.

Activity 2: Muscles & Fascia



2.1.1
Proximal: ASIS, anterior part of iliac crest
Distal : Iliotibial trac, which attaches to lateral condyle of tibia.
2.1.2
Gluteus maximus.
2.1.3
Superior gluteal nerve( L5,S1)

2.2.1
Trochanteric bursa , the ischial bursa and gluteofemoral bursa.

2.2.2
Separate the gluteus maximus from adjacent structures, reduce friction and permit free movement.

2.2.3
A type of friction bursitis resulting from excessive friction between the ischial bursa and ischial tuberosities.

2.2.3.1
Recurrent mcirotrauma resulting from repeated stress eg : cycling, rowing or other activities involving hip extension while seated.


2.3.1
Superior gluteal nerve
2.3.2
Abduct and medially rotate thigh: keep pelvis level when ipsilateral limb is weight-bearing and advance opposite side during swing phase.

Activity 3: Vessels
3.1
Superior and inferior gluteal arteries.
3.2
Internal iliac artery which is a branch of the common iliac artery
3.2.1


Activity 4: Nerves
4.1 Inferior gluteal nerve
4.1.1
Compression and ischemia in sedentary individuals.
4.1.2
Difficulty in rising from seated position and climbing stairs.

4.2
Injections into the buttock are safe only in the superolateral quadrant of the buttock or superior to a line extending from the PSIS to the superior border of the greater trochanter (approximating the superior border of the gluteus maximus). IM injections can also be given safely into the anterolateral part of the thigh, where the needle enters the tensor fasciae latae as it extends distally from the iliac crest and ASIS. The index finger is placed on the ASIS, and the fingers are spread posteriorly along the iliac crest until the tubercle of the crest is felt by the middle finger . An IM injection can be made safely in the triangular area between the fingers (just anterior to the proximal joint of the middle finger) because it is superior to the sciatic nerve.